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Breathing and Exchange of Gases

Breathing and Exchange of GasesNEET Zoology · Class 11 · NCERT Chapter 14

3 interactive concept widgets for Breathing and Exchange of Gases. Drag any slider, change any number, and watch the formula and the answer update live. Built so you understand how each NEET problem actually works, not just the final number.

Respiratory system explorer

Click any part of the human respiratory tract to see its role and the NEET fact tested about it.

Respiratory system anatomy

Human respiratory system: click any labelled part

A labelled diagram of the human respiratory system. Click any pin or chip to see what each part does and the key NEET fact about it.

DIAPHRAGM
Nasal Chamber
Pharynx
Larynx (Voice Box)
Trachea (Windpipe)
Bronchi
Bronchioles
Alveoli
Lungs
Diaphragm

Trachea (Windpipe)

About 11 to 12 cm long. Kept open by C-shaped cartilaginous rings (the open side faces the oesophagus). Lined with ciliated mucus epithelium that sweeps debris upward. Divides at the carina into two primary bronchi.

NEET fact

Trachea has C-shaped cartilaginous rings. The right primary bronchus is wider, shorter and more vertical, so foreign objects more often lodge there.

Try this

  • Click the larynx. What is the difference between the glottis and the epiglottis? Which one closes when you swallow?
  • Compare the bronchi to the bronchioles. Which one has cartilage? What does the absence of cartilage in bronchioles mean for asthma?
  • Click the alveoli. Note the type I and type II pneumocytes. Which type secretes surfactant, and why does surfactant matter?

Mechanism of breathing

Toggle between inspiration and expiration to see the diaphragm, intercostal muscles, thoracic volume and pressure changes.

Breathing mechanism

Mechanism of breathing: inspiration vs expiration

Toggle between inspiration and expiration to see how the diaphragm, intercostal muscles, thoracic volume, and air pressure change. A side-by-side comparison table shows all changes at a glance.

Inspiration (Inhalation)
Expiration (Exhalation)
DIAPHRAGMAIR INribsup+outribsup+out

Inspiration (Inhalation)

Both the diaphragm and external intercostal muscles contract simultaneously. The thoracic cavity expands. Because the lungs are attached to the chest wall via the pleura, they expand too. The increased volume lowers the air pressure inside the lungs below atmospheric pressure, so air flows in.

NEET fact

Inspiration is an ACTIVE process: muscles must contract. Quiet expiration is largely PASSIVE (muscles relax).

Feature

Inspiration

Expiration

Diaphragm

Contracts and flattens downward

Relaxes and domes upward

External intercostals

Contract: ribs pulled upward and outward

Relax: ribs fall back

Internal intercostals

Relaxed

Contract in FORCED expiration (e.g. coughing, exercise)

Thoracic cavity volume

Increases (both vertical and lateral dimensions increase)

Decreases

Intra-pulmonary pressure

Falls to about 1 to 3 mm Hg below atmospheric pressure

Rises to about 1 to 3 mm Hg above atmospheric pressure

Air movement

Air rushes IN (from high pressure outside to lower pressure inside)

Air is pushed OUT (from higher pressure inside to atmospheric outside)

Try this

  • Toggle between inspiration and expiration. Watch the diaphragm position and lung size change in the diagram.
  • In quiet breathing, which phase is active (requires muscle contraction) and which is passive? Try to state why before clicking.
  • When would forced expiration be used? What muscles are added to quiet expiration during exercise or coughing?

Oxygen dissociation curve

Shift the oxygen-haemoglobin dissociation curve with pCO2, pH and temperature to see the Bohr effect.

Gas transport

Oxygen-haemoglobin dissociation curve: explore the Bohr effect

An interactive sigmoid O2 dissociation curve. Use the sliders to change pCO2, pH and temperature and watch the Bohr effect shift the curve left or right. See how much O2 is loaded in the lungs and delivered to the tissues.

pO2 (mm Hg)% Saturation20406080100255075100NormalLung (97%)Tissue (75%)P50=26.5

pCO2: 40 mm Hg

20 (low)

70 (high)

pH: 7.4

7.1 (acid)

7.7 (alk)

Temperature: 37 °C

30 (cold)

43 (fever)

Normal (P50 = 26.5 mm Hg)

Normal physiological conditions (pH 7.4, pCO2 40 mm Hg, 37 C)

Sat at Lung (pO2 100)

97.3%

Sat at Tissue (pO2 40)

75.2%

O2 Delivered

22.1%

NEET key facts

!

The dissociation curve is S-shaped (sigmoid) due to cooperative binding of O2 to haemoglobin.

!

P50 = the pO2 at which haemoglobin is 50% saturated. Normal value ~26.5 mm Hg.

!

Right shift (higher P50): caused by high pCO2, low pH, or high temperature. Haemoglobin releases more O2.

!

Left shift (lower P50): caused by low pCO2, high pH, or low temperature. Haemoglobin picks up O2 more readily.

!

The Bohr effect means active tissues (high CO2, low pH, high temp) automatically receive more O2.

!

97% of O2 is transported as oxyhaemoglobin; only 3% is dissolved in plasma.

Try this

  • Set pCO2 to 70 mm Hg (as in exercising muscle). Does the curve shift left or right? Does haemoglobin hold onto O2 more tightly or release it more readily?
  • Now reset and lower pH to 7.1. This mimics acidosis. How does the O2 delivery figure change compared to normal?
  • Set temperature to 43 C (high fever). Which direction does the curve shift? What does this mean for O2 delivery to hot tissues?

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